Payne Stewart had ADD; PCB exposed? ATSDR Report

[GCJeffrey@aol.com]

Golfer Payne Stewart's mother stated last night
on Inside Edition that Payne had Attention
Deficit Disorder. 

(Did his mother play golf on a contaimated
golf course? eat fish?)

This is another item that you can use in your
efforts to eliminate dioxin production and
real clean up of PCBs down to below 1 part per
trillion.


George C. Jeffrey BS Chemical Engineering
Citizens Reaction Against Pollution

I Internet posted the unedited ATSDR literature
review on PCBs linked to ADD/ADHD on our site.
The links are on both the homepage and
on the Briefing Book Page.
<A HREF="http://sites.netscape.net/georgecjeffrey/homepage">http://sites.netsc
ape.net/georgecjeffrey/homepage</A> 
<A HREF="http://sites.netscape.net/GeorgeCJeffrey/briefingbookattach">http://s
ites.netscape.net/GeorgeCJeffrey/briefingbookattach</A> 


HOW TO USE THIS ATSDR INFORMATION:

Below are excerts of an ongoing debate
with "Joel" on the Times Union newspaper forum.

The ATSDR PCB Literature Summary Follows.

============================
Comments about stories published 10/26/99. 
Payne Stewart.....Corporate America Supported Hero
Posted by: George C. Jeffrey (205.188.192.153)
Posted on: 10/27/99 - 09:13 a.m.
Joel
RJ

Pardon,
I should not have insulted your religion: GOLF!!!!
......
For you information, Payne Stewart had ATTENTION
DEFICIT DISORDER (ADD).
(Confirmed on Inside Edition last night by his mother).

Many children and some adults take Ritatin (tm) for ADD.

According to Dr. Mary Ann Block, Ritalin affect the
brain in the same way as COCAINE.
(Book, "No more Ritalin," usually on the shelf
in larger bookstores).

You are living in Jacksonville Florida which has
one of the highest ADD/ADHD-Ritalin rates in
the world ..... and has one of the lowest US
graduation rates for high school: 48%.

ADD/ADHD is stongly linked to toxic PCB exposure
(found in older florescent light fixture and other
electrical equipment).
.....
.....
(To: Joel)
 Here are the type of facts which you avoid:

Exposure to PCB's (and golf course chemicals)
can lead to:
"Intellectual and behavioral abnormalities,
including attention deficit hyperactivity disorder"


ATSDR Literature Review on PCBs-Dioxin:
==============================
PUBLIC HEALTH IMPLICATIONS
OF EXPOSURE TO
POLYCHLORINATED BIPHENYLS (PCBs) 
http://www.atsdr.cdc.gov/DT/pcb007.html

Barry L. Johnson, Heraline E. Hicks, William
Cibulas, Obaid Faroon, Annette E. Ashizawa,
Christopher T. De Rosa 
Agency for Toxic Substances and Disease Registry 
Public Health Service 
U.S. Department of Health and Human Services 
and 
Vincent J. Cogliano, Milton Clark 
U.S. Environmental Protection Agency

ABSTRACT 

This paper summarizes the health implications
associated with exposure to polychlorinated
biphenyls (PCBs), primarily through fish
consumption.

Recent studies complement and add to the
scientific data gathered over the last two
decades that document health consequences
associated with exposures to PCBs.

Although much of the research has been
undertaken in the Great Lakes basin, the health
implications are international.

The findings of elevated PCB levels in human
populations, together with findings of
developmental deficits and neurologic
problems in children whose mothers ate
PCB-contaminated fish, have compelling
implications.

The weight of evidence clearly indicates that
populations continue to eat fish containing
PCBs and that significant health consequences
are associated with consumption of large
amounts of some fish.

Although PCBs are declining in the
environment, health concerns are still
warranted.


Recent findings indicate that susceptible
populations (e.g., certain ethnic groups, sport
anglers, the elderly, pregnant women, children,
fetuses, and nursing infants) continue to be
exposed to PCBs via fish and wildlife
consumption.

Human health studies discussed in this
summary indicate that

(1) reproductive function may be disrupted
by exposure to PCBs;

(2) neurobehavioral and developmental deficits
occur in newborns and continue through
school-aged children who had in utero
exposure to PCBs;

(3) other systemic effects (e.g., self-reported
liver disease and diabetes, and effects on the
thyroid and immune systems) are associated
with elevated serum levels of PCBs;

and

(4) increased cancer risks, e.g., non-Hodgkin's
lymphoma, are associated with PCB exposures.



Resistance to thyroid hormone:
implications for neurodevelopmental
research on the effects of thyroid hormone disruptors.

Hauser P, McMillin JM, Bhatara VS
Department of Psychiatry,
University of Maryland Medical Center,
Baltimore, USA. 
Hauser.Peter@Baltimore.va.gov
Toxicol Ind Health 1998 Jan-Apr;14(1-2):85-101 

Thyroid hormones are essential for normal
behavioral, intellectual, and neurological
development.

Congenital hypothyroidism, if not treated, can
result in irreversible mental retardation,
whereas thyroid diseases with more moderate
impairment of thyroid function, such as
resistance to thyroid hormone, cause less
severe intellectual and behavioral
abnormalities, including attention deficit
hyperactivity disorder.

There is increasing evidence that exposure to
certain synthetic compounds, including dioxins
and polychlorinated biphenyls (PCBs), during
the perinatal period can also impair learning,
memory, and attentional processes in offspring.

Animal and human studies suggest that
exposure to these environmental toxicants
impair normal thyroid function.

Although the precise mechanisms of action of
the adverse effects these toxicants have on
neurodevelopment have not yet been
elucidated, it is possible that they are partially
or predominantly mediated by alterations in
hormone binding to the thyroid hormone
receptor.

The convergence of studies that examine the
neurodevelopmental consequences of moderate
impairment of thyroid function, such as is
found in resistance to thyroid hormone, with
those studies that demonstrate the adverse
behavioral and cognitive effects of perinatal
exposure to dioxins and PCBs serves to
generate new hypotheses to test in a research
setting.

Such studies may provide new insights into
the basic pathogenesis of developmental
neurotoxicity following exposure to
thyroid-disrupting synthetic compounds.

Publication Types:
Review 
Review, tutorial 
PMID: 9460171, UI: 98121473 

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